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In addition, insulin from MIF-deficient mice had another conformation or posttranslational modification when compared to insulin from wildtype mice. MIF actually stimulated insulin hexamer formation in cell-cost-free devices [100]. Thus, it seems that MIF can act as an adaptor protein for insulin oligomerization. Insulin was also uncovered to co-im

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MIF was demonstrated to rescue neurons from oxidative strain induced apoptosis by inhibiting caspase-3 activation, and MIF-knockout mice also experienced far more lifeless neurons, in addition to a higher infarct sizing just after induction of an experimental stroke [124]. presented these upstream steps in the inflammatory cascade, it is not stunn

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